Neuronal vulnerability of CLN3 deletion to calcium-induced cytotoxicity is mediated by calsenilin-Reference-Cited by-同舟云学术

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Neuronal vulnerability of CLN3 deletion to calcium-induced cytotoxicity is mediated by calsenilin

Published:2006-12-22 Issue:3 Volume:16 Page:317-326
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Chang Jae-Woong,Choi Hyunwoo,Kim Hyun-Ji,Jo Dong-Gyu,Jeon Young-Jun,Noh Jee-Yeon,Park Woo Jin,Jung Yong-Keun

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/16/3/317/17244187/ddl466.pdf

Reference46 articles.

1. Calasenilin, a calcium-binding protein that interacts with the presenilins and regulates the levels of a presenilin fragment;Buxbaum;Nat. Med.,1998

2. DREAM is a Ca2+-regulated transcriptional repressor;Carrion;Nature,1999

3. Modulation of A-type potassium channels by a family of calcium sensors;An;Nature,2000

4. Pro-apoptotic function of calsenilin/DREAM/KChIP3;Jo;FASEB J.,2001

5. Induction of pro-apoptotic calsenilin/DREAM/KChIP3 in Alzheimer's disease and cultured neurons after amyloid-β exposure;Jo;J. Neurochem.,2004

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