Arabidopsis UGT76B1 glycosylates N-hydroxy-pipecolic acid and inactivates systemic acquired resistance in tomato
Author:
Affiliation:
1. Department of Chemical Engineering, Stanford University, Stanford, CA 94305, USA
2. Department of Biology, Stanford University, Stanford, CA 94305, USA
3. Howard Hughes Medical Institute, Stanford University, Stanford, CA 94305, USA
Abstract
Funder
Howard Hughes Medical Institute-Simons Faculty Scholar award
National Science Foundation Graduate Research Fellowship
National Science Foundation
inational Science Foundation
Ministry of Science and Technology of Taiwan
Publisher
Oxford University Press (OUP)
Subject
Cell Biology,Plant Science
Link
http://academic.oup.com/plcell/advance-article-pdf/doi/10.1093/plcell/koaa052/36349195/koaa052.pdf
Reference52 articles.
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3. The isoleucic acid triad: distinct impacts on plant defense, root growth, and formation of reactive oxygen species;Bauer;J Exp Bot,2020
4. UGT76B1, a promiscuous hub of small molecule-based immune signaling, glucosylates N-hydroxypipecolic acid and controls basal pathogen defense;Bauer;Plant Cell,2020
5. Pipecolic acid orchestrates plant systemic acquired resistance and defense priming via salicylic acid-dependent and -independent pathways;Bernsdorff;Plant Cell,2016
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