Plasma kidney injury molecule-1 (p-KIM-1) levels and deterioration of kidney function over 16 years
Author:
Affiliation:
1. Department of Clinical Sciences, Lund University, Malmö, Sweden
2. Department of Nephrology, Lund University, Malmö, Sweden
3. Department of Medicine, Lund University, Malmö, Sweden
Abstract
Funder
European Research Council
Swedish Research Council
Swedish Heart and Lung Foundation
Novo Nordic Foundation
Swedish Diabetes Foundation
Påhlsson Foundation
Knut and Alice Wallenberg Foundation
Region Skåne
Skåne University Hospital
Linneus Foundation for the Lund University Diabetes Center and Swedish Foundation
Strategic Research
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/35/2/265/32739599/gfy382.pdf
Reference30 articles.
1. Kidney injury molecule-1 (KIM-1), a putative epithelial cell adhesion molecule containing a novel immunoglobulin domain, is up-regulated in renal cells after injury;Ichimura;J Biol Chem,1998
2. Chronic epithelial kidney injury molecule-1 expression causes murine kidney fibrosis;Humphreys;J Clin Invest,2013
3. Kidney injury molecule-1 (KIM-1): a novel biomarker for human renal proximal tubule injury;Han;Kidney Int,2002
4. Tubular markers do not predict the decline in glomerular filtration rate in type 1 diabetic patients with overt nephropathy;Nielsen;Kidney Int,2011
5. Tubular markers are associated with decline in kidney function in proteinuric type 2 diabetic patients;Nielsen;Diabetes Res Clin Pract,2012
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