Myofibroblast transdifferentiation of mesothelial cells is mediated by RAGE and contributes to peritoneal fibrosis in uraemia
Author:
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/21/9/2549/7624860/gfl271.pdf
Reference20 articles.
1. Williams JD, Craig KJ, Topley N et al. Morphologic changes in the peritoneal membrane of patients with renal disease. J Am Soc Nephrol2002; 13: 470–479
2. Williams JD, Craig KJ, Von Ruhland C et al. The natural course of peritoneal membrane biology during peritoneal dialysis. Kidney Int2003; 88 [Suppl]: S43–S49
3. Miyata T, Devuyst O, Kurokawa K, van Ypersele dS. Toward better dialysis compatibility: advances in the biochemistry and pathophysiology of the peritoneal membranes. Kidney Int2002; 61: 375–386
4. Nakayama M, Kawaguchi Y, Yamada K et al. Immunohistochemical detection of advanced glycosylation end-products in the peritoneum and its possible pathophysiological role in CAPD. Kidney Int1997; 51: 182–186
5. Honda K, Nitta K, Horita S et al. Accumulation of advanced glycation end products in the peritoneal vasculature of continuous ambulatory peritoneal dialysis patients with low ultra-filtration. Nephrol Dial Transplant1999; 14: 1541–1549
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