Deregulation of SATB2 in carcinogenesis with emphasis on miRNA-mediated control
Author:
Affiliation:
1. Department of Environmental Medicine, New York University School of Medicine, New York, NY, USA
Funder
NIH
Publisher
Oxford University Press (OUP)
Subject
Cancer Research,General Medicine
Link
http://academic.oup.com/carcin/article-pdf/40/3/393/28639445/bgz020.pdf
Reference111 articles.
1. Isolation and characterization of SATB2, a novel AT-rich DNA binding protein expressed in development- and cell-specific manner in the rat brain;Szemes;Neurochem. Res,2006
2. Identification of SATB2 as the cleft palate gene on 2q32-q33;FitzPatrick;Hum. Mol. Genet,2003
3. The role of SATB2 in skeletogenesis and human disease;Zhao;Cytokine Growth Factor Rev,2014
4. Clinical and molecular consequences of disease-associated de novo mutations in SATB2;Bengani;Genet. Med,2017
5. Novel transcription factor Satb2 interacts with matrix attachment region DNA elements in a tissue-specific manner and demonstrates cell-type-dependent expression in the developing mouse CNS;Britanova;Eur. J. Neurosci,2005
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