SMAD7 in keratinocytes promotes skin carcinogenesis by activating ATM-dependent DNA repair and an EGFR-mediated cell proliferation pathway-Reference-Cited by-同舟云学术

SMAD7 in keratinocytes promotes skin carcinogenesis by activating ATM-dependent DNA repair and an EGFR-mediated cell proliferation pathway

Published:2018-09-14 Issue:1 Volume:40 Page:112-120
ISSN:0143-3334
Container-title:Carcinogenesis
language:en
Short-container-title:

Author:

Ha Thi Huyen Trang¹,Kim Hye-Youn¹,Lee Young-Jae²,Kim Seong-Jin³,Hong Suntaek¹^ORCID

Affiliation:

1. Laboratory of Cancer Cell Biology, Department of Biochemistry, Gachon University School of Medicine, Incheon, Republic of Korea

2. Laboratory of Developmental Genetics, Department of Biochemistry, Gachon University School of Medicine, Incheon, Republic of Korea

3. Department of Transdisciplinary Studies, Graduate School of Convergence Science and Technology, Seoul National University, Suwon, Republic of Korea

Funder

National Research Foundation of Korea

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,General Medicine

Link

http://academic.oup.com/carcin/article-pdf/40/1/112/28044542/bgy121.pdf

Reference32 articles.

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4. A functional role for Smad7 in sustaining colon cancer cell growth and survival;Stolfi;Cell Death Dis,2014

5. Increased risk of colon cancer associated with a genetic polymorphism of SMAD7;Slattery;Cancer Res,2010

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