(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells-Reference-Cited by-同舟云学术

(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells

Published:2012-11-24 Issue:3 Volume:34 Page:627-637
ISSN:0143-3334
Container-title:Carcinogenesis
language:en
Short-container-title:Carcinogenesis

Author:

Liu S.,Li H.,Chen L.,Yang L.,Li L.,Tao Y.,Li W.,Li Z.,Liu H.,Tang M.,Bode A. M.,Dong Z.,Cao Y.

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,General Medicine

Link

http://academic.oup.com/carcin/article-pdf/34/3/627/7452821/bgs364.pdf

Reference54 articles.

1. Clonal Proliferations of Cells Infected with Epstein–Barr Virus in Preinvasive Lesions Related to Nasopharyngeal Carcinoma

2. Detection of Epstein-Barr Virus DNA in Hepatocellular Carcinoma Tissues from Hepatitis C-positive Patients

3. SPECTRUM OF EPSTEIN-BARR VIRUS–ASSOCIATED DISEASES

4. Quantitative analysis of circulating cell-free Epstein-Barr virus (EBV) DNA levels in patients with EBV-associated lymphoid malignancies

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