Oestrogen fuels the growth of endometrial hyperplastic lesions initiated by overactive Wnt/β-catenin signalling
Author:
Affiliation:
1. Gynaecology Oncology Group, School of Biomedical Sciences and Pharmacy, University of Newcastle, Callaghan, New South Wales Australia
Funder
National Health and Medical Research Council
Australian Research Council
Cancer Institute NSW
Publisher
Oxford University Press (OUP)
Subject
Cancer Research,General Medicine
Link
http://academic.oup.com/carcin/article-pdf/39/9/1105/25820952/bgy079.pdf
Reference44 articles.
1. Cancer incidence and mortality worldwide: sources, methods and major patterns in GLOBOCAN 2012;Ferlay;Int. J. Cancer,2015
2. Endometrial cancer;Amant;Lancet,2005
3. Two pathogenetic types of endometrial carcinoma;Bokhman;Gynecol. Oncol,1983
4. Endometrial carcinoma;Di Cristofano;Annu. Rev. Pathol,2007
5. Molecular profiling of endometrial malignancies;Samarnthai;Obstet. Gynecol. Int,2010
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2. Role of non‑coding RNA intertwined with the Wnt/β‑catenin signaling pathway in endometrial cancer (Review);Molecular Medicine Reports;2023-06-20
3. D-Chiro-Inositol in Endometrial Hyperplasia: A Pilot Study;International Journal of Molecular Sciences;2023-06-13
4. The interplay of sex steroid hormones and microRNAs in endometrial cancer: current understanding and future directions;Frontiers in Endocrinology;2023-04-21
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