Genetic versus chemoprotective activation of Nrf2 signaling: overlapping yet distinct gene expression profiles between Keap1 knockout and triterpenoid-treated mice-Reference-Cited by-同舟云学术

Genetic versus chemoprotective activation of Nrf2 signaling: overlapping yet distinct gene expression profiles between Keap1 knockout and triterpenoid-treated mice

Published:2009-04-21 Issue:6 Volume:30 Page:1024-1031
ISSN:1460-2180
Container-title:Carcinogenesis
language:en
Short-container-title:

Author:

Yates Melinda S.,Tran Quynh T.,Dolan Patrick M.,Osburn William O.,Shin Soona,McCulloch Colin C.,Silkworth Jay B.,Taguchi Keiko,Yamamoto Masayuki,Williams Charlotte R.,Liby Karen T.,Sporn Michael B.,Sutter Thomas R.,Kensler Thomas W.

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,General Medicine

Link

http://academic.oup.com/carcin/article-pdf/30/6/1024/17287371/bgp100.pdf

Reference48 articles.

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2. Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants;Dinkova-Kostova;Proc. Natl Acad. Sci. USA,2002

3. Modulation of gene expression by cancer chemopreventive dithiolethiones through the Keap1-Nrf2 pathway. Identification of novel gene clusters for cell survival;Kwak;J. Biol. Chem.,2003

4. High sensitivity of Nrf2 knockout mice to acetaminophen hepatotoxicity associated with decreased expression of ARE-regulated drug metabolizing enzymes and antioxidant genes;Enomoto;Toxicol. Sci.,2001

5. In vivo modulation of the Parkinsonian phenotype by Nrf2;Burton;Neurotoxicology,2006

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