Inducible Nitric Oxide Synthase (iNOS) and Nitric Oxide (NO) are Important Mediators of Reflux-induced Cell Signalling in Esophageal Cells-Reference-Cited by-同舟云学术

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Inducible Nitric Oxide Synthase (iNOS) and Nitric Oxide (NO) are Important Mediators of Reflux-induced Cell Signalling in Esophageal Cells

Published:2012-07-23 Issue:11 Volume:33 Page:2035-2043
ISSN:0143-3334
Container-title:Carcinogenesis
language:en
Short-container-title:Carcinogenesis

Author:

McAdam E.,Haboubi H. N.,Forrester G.,Eltahir Z.,Spencer-Harty S.,Davies C.,Griffiths A. P.,Baxter J. N.,Jenkins G. J. S.

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,General Medicine

Link

http://academic.oup.com/carcin/article-pdf/33/11/2035/7460465/bgs241.pdf

Reference59 articles.

1. Risk of Malignant Progression in Barrett's Esophagus Patients: Results from a Large Population-Based Study

2. Tumor Necrosis Factor and Granulocyte Macrophage-Colony Stimulating Factor Stimulate Human Macrophages to Restrict Growth of Virulent Mycobacterium avium and to Kill Avirulent M. avium: Killing Effector Mechanism Depends on the Generation of Reactive Nitr

3. Activation of human macrophages for the killing of intracellular Trypanosoma cruzi by TNF-α and IFN-γ through a nitric oxide-dependent mechanism

4. The killing of Leishmania major by human macrophages is mediated by nitric oxide induced after ligation of the Fc epsilon RII/CD23 surface antigen.

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