Decorin-mediated inhibition of colorectal cancer growth and migration is associated with E-cadherin in vitro and in mice-Reference-Cited by-同舟云学术

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Decorin-mediated inhibition of colorectal cancer growth and migration is associated with E-cadherin in vitro and in mice

Published:2011-12-08 Issue:2 Volume:33 Page:326-330
ISSN:0143-3334
Container-title:Carcinogenesis
language:en
Short-container-title:Carcinogenesis

Author:

Bi X.,Pohl N. M.,Qian Z.,Yang G. R.,Gou Y.,Guzman G.,Kajdacsy-Balla A.,Iozzo R. V.,Yang W.

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,General Medicine

Link

http://academic.oup.com/carcin/article-pdf/33/2/326/7460316/bgr293.pdf

Reference43 articles.

1. Proteoglycans in health and disease: novel regulatory signaling mechanisms evoked by the small leucine-rich proteoglycans

2. Tumor microenvironment: Modulation by decorin and related molecules harboring leucine-rich tandem motifs

3. Genetic deficiency of decorin causes intestinal tumor formation through disruption of intestinal cell maturation

4. De novo decorin gene expression suppresses the malignant phenotype in human colon cancer cells.

5. Transforming growth factor-β and p-21: multiple molecular targets of decorin-mediated suppression of neoplastic growth

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1. Proteoglycans of basement membranes: Crucial controllers of angiogenesis, neurogenesis, and autophagy;Proteoglycan Research;2024-06-29

2. Characterisation of colorectal cancer by hierarchical clustering analyses for five stroma‐related markers;The Journal of Pathology: Clinical Research;2024-06-18

3. Mechanisms Underlying the Rarity of Skeletal Muscle Cancers;International Journal of Molecular Sciences;2024-06-12

4. Decorin suppresses stemness and migration potential of malignant peripheral nerve sheath tumor through inhibiting epidermal growth factor receptor signaling;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-06

5. Decorin (DCN) Downregulation Activates Breast Stromal Fibroblasts and Promotes Their Pro-Carcinogenic Effects through the IL-6/STAT3/AUF1 Signaling;Cells;2024-04-14

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