Inhibition of apoptosis by downregulation of hBex1, a novel mechanism, contributes to the chemoresistance of Bcr/Abl+ leukemic cells
Author:
Publisher
Oxford University Press (OUP)
Subject
Cancer Research,General Medicine
Link
http://academic.oup.com/carcin/article-pdf/30/1/35/906271/bgn251.pdf
Reference36 articles.
1. A requirement for NF-kappa B activation in Bcr-Abl-mediated transformation
2. BCR-ABL-mediated inhibition of apoptosis with delay of G2/M transition after DNA damage: a mechanism of resistance to multiple anticancer agents
3. Phosphatidylinositol-3 kinase activity is regulated by BCR/ABL and is required for the growth of Philadelphia chromosome-positive cells
4. Synergistic activity of imatinib and 17-AAG in imatinib-resistant CML cells overexpressing BCR-ABL – Inhibition of P-glycoprotein function by 17-AAG
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