Sodium current deficit and arrhythmogenesis in a murine model of plakophilin-2 haploinsufficiency-Reference-Cited by-同舟云学术

Sodium current deficit and arrhythmogenesis in a murine model of plakophilin-2 haploinsufficiency

Published:2012-07-03 Issue:4 Volume:95 Page:460-468
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Cerrone Marina,Noorman Maartje,Lin Xianming,Chkourko Halina,Liang Feng-Xia,van der Nagel Roel,Hund Thomas,Birchmeier Walter,Mohler Peter,van Veen Toon A.,van Rijen Harold V.,Delmar Mario

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/95/4/460/17200332/cvs218.pdf

Reference44 articles.

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2. Tyrosine-phosphorylated and nonphosphorylated sodium channel beta1 subunits are differentially localized in cardiac myocytes;Malhotra;J Biol Chem,2004

3. Interactions between ankyrin-G, plakophilin-2, and connexin43 at the cardiac intercalated disc;Sato;Circ Res,2011

4. Loss of plakophilin-2 expression leads to decreased sodium current and slower conduction velocity in cultured cardiac myocytes;Sato;Circ Res,2009

5. The cardiac desmosome and arrhythmogenic cardiomyopathies: from gene to disease;Delmar;Circ Res,2010

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