Tau deposition drives neuropathological, inflammatory and behavioral abnormalities independently of neuronal loss in a novel mouse model-Reference-Cited by-同舟云学术

Tau deposition drives neuropathological, inflammatory and behavioral abnormalities independently of neuronal loss in a novel mouse model

Published:2015-08-13 Issue:21 Volume:24 Page:6198-6212
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Hum. Mol. Genet.

Author:

Cook Casey,Kang Silvia S.,Carlomagno Yari,Lin Wen-Lang,Yue Mei,Kurti Aishe,Shinohara Mitsuru,Jansen-West Karen,Perkerson Emilie,Castanedes-Casey Monica,Rousseau Linda,Phillips Virginia,Bu Guojun,Dickson Dennis W.,Petrucelli Leonard,Fryer John D.

Funder

Mayo Clinic Foundation

National Institutes of Health/National Institute on Aging

National Institutes of Health/National Institute of Neurological Disorders and Stroke

BrightFocus Foundation

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/24/21/6198/1672623/ddv336.pdf

Reference74 articles.

1. Association of missense and 5′-splice-site mutations in tau with the inherited dementia FTDP-17

2. Tau is a candidate gene for chromosome 17 frontotemporal dementia

3. Mutation in the tau gene in familial multiple system tauopathy with presenile dementia

4. Novel mutation in MAPT exon 13 (p.N410H) causes corticobasal degeneration

5. Genome-wide association study reveals genetic risk underlying Parkinson's disease

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