Adolescent Synthetic Cannabinoid Exposure Produces Enduring Changes in Dopamine Neuron Activity in a Rodent Model of Schizophrenia Susceptibility

Author:

Aguilar David D1ORCID,Giuffrida Andrea1,Lodge Daniel J1

Affiliation:

1. Department of Pharmacology and Center for Biomedical Neuroscience, University of Texas Health Science Center at San Antonio, Texas

Funder

National Institute of Mental Health

National Institutes of Health

Publisher

Oxford University Press (OUP)

Subject

Pharmacology (medical),Psychiatry and Mental health,Pharmacology

Reference68 articles.

1. Increasing endocannabinoid levels in the ventral pallidum restore aberrant dopamine neuron activity in the subchronic PCP rodent model of schizophrenia;Aguilar;Int J Neuropsychopharmacol,2014

2. THC and endocannabinoids differentially regulate neuronal activity in the prefrontal cortex and hippocampus in the subchronic PCP model of schizophrenia;Aguilar;J Psychopharmacol,2016

3. Cannabis use in adolescence and risk for adult psychosis: longitudinal prospective study;Arseneault;BMJ,2002

4. Causal association between cannabis and psychosis: examination of the evidence;Arseneault;Br J Psychiatry,2004

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