Metformin attenuates susceptibility to inflammation-induced preterm birth in mice with higher endocannabinoid levels†-Reference-Cited by-同舟云学术

Metformin attenuates susceptibility to inflammation-induced preterm birth in mice with higher endocannabinoid levels†

Published:2017-12-05 Issue:2 Volume:98 Page:208-217
ISSN:0006-3363
Container-title:Biology of Reproduction
language:en
Short-container-title:

Author:

Sun Xiaofei¹,Tavenier Alexandra¹,Deng Wenbo¹,Leishman Emma²,Bradshaw Heather B²,Dey Sudhansu K¹

Affiliation:

1. Division of Reproductive Sciences, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, USA

2. Department of Psychological and Brain Sciences, Kinsey Institute for Research in Sex, Gender, and Reproduction, Indiana University, Bloomington, Indiana, USA

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,General Medicine,Reproductive Medicine

Link

http://academic.oup.com/biolreprod/article-pdf/98/2/208/24012326/iox164.pdf

Reference63 articles.

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2. Heightened uterine mammalian target of rapamycin complex 1 (mTORC1) signaling provokes preterm birth in mice;Hirota;Proc Natl Acad Sci USA,2011

3. Sustained endocannabinoid signaling compromises decidual function and promotes inflammation-induced preterm birth;Sun;J Biol Chem,2016

4. Isolation and structure of a brain constituent that binds to the cannabinoid receptor;Devane;Science,1992

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