Influence of the genetic background on the reproductive phenotype of mice lacking Cysteine-Rich Secretory Protein 1 (CRISP1)†

Author:

Weigel Muñoz Mariana1,Battistone María A1,Carvajal Guillermo1,Maldera Julieta A1,Curci Ludmila1,Torres Pablo2,Lombardo Daniel2,Pignataro Omar P13,Da Ros Vanina G1,Cuasnicú Patricia S1

Affiliation:

1. Instituto de Biología y Medicina Experimental (IByME-CONICET), Buenos Aires, Argentina

2. Instituto de Investigación y Tecnología en Reproducción Animal, Facultad de Ciencias Veterinarias, Universidad de Buenos Aires, Buenos Aires, Argentina

3. Departamento de Química Biológica, Facultad de Ciencias Exactas y Naturales, Universidad de Buenos Aires, Buenos Aires, Argentina

Funder

World Health Organization

National Research Council

National Agency for Scientific and Technological Promotion

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,General Medicine,Reproductive Medicine

Reference67 articles.

1. Capacitation of mouse spermatozoa. I. Correlation between the capacitation state and protein tyrosine phosphorylation;Visconti;Development,1995

2. Capacitation of mouse spermatozoa. II. Protein tyrosine phosphorylation and capacitation are regulated by a cAMP-dependent pathway;Visconti;Development,1995

3. Ca2+ uptake during capacitation of mouse spermatozoa and the effect of an anion transport inhibitor on Ca2+ uptake;Ruknudin;Mol Reprod Dev,1990

4. From the epididymis to the egg: participation of CRISP proteins in mammalian fertilization;Da Ros;Asian J Androl,2015

5. The CAP superfamily: cysteine-rich secretory proteins, antigen 5, and pathogenesis-related 1 proteins—roles in reproduction, cancer, and immune defense;Gibbs;Endocr Rev,2008

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