Why, when and how should exposure be considered at the within-host scale? A modelling contribution to PRRSv infection

Author:

Go Natacha12,Belloc Catherine1,Bidot Caroline2,Touzeau Suzanne34

Affiliation:

1. BIOEPAR, INRA, Oniris, LUNAM Université, Nantes, France

2. MaIAGE, INRA, Université Paris-Saclay, Jouy-en-Josas, France

3. ISA, INRA, CNRS, Université Côte d’Azur, France

4. BIOCORE, Inria, INRA, CNRS, UPMC Université, Université Côte d’Azur, France

Publisher

Oxford University Press (OUP)

Subject

Applied Mathematics,Pharmacology,General Environmental Science,General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Modeling and Simulation,General Medicine,General Neuroscience

Reference57 articles.

1. Host inhibits replication of European porcine reproductive and respiratory syndrome virus in macrophages by altering differential regulation of type-I interferon transcriptional responses;Ait-Ali;Immunogenetics,2011

2. A review of mathematical models of influenza A infections within a host or cell culture: lessons learned and challenges ahead;Beauchemin;BMC Public Health,2011

3. Pathogen dose infectivity curves as a method to analyze the distribution of host susceptibility: a quantitative assessment of maternal effects after food stress and pathogen exposure;Ben-Ami;Am. Nat.,2010

4. Borghetti, P. (2005) Cell-mediated immunity and viral infection in pig. PRRS fatti vs speculazioni. Università degli Studi di Parma, Dipartimento di Salute Animale. Parma, Italy. pp. 27–46.

5. Viral and bacterial interactions in the upper respiratory tract;Bosch;PLoS Pathog,2013

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