Genome-wide association study in multiple human prion diseases suggests genetic risk factors additional to PRNP-Reference-Cited by-同舟云学术

Genome-wide association study in multiple human prion diseases suggests genetic risk factors additional to PRNP

Published:2011-12-30 Issue:8 Volume:21 Page:1897-1906
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Mead Simon,Uphill James,Beck John,Poulter Mark,Campbell Tracy,Lowe Jessica,Adamson Gary,Hummerich Holger,Klopp Norman,Rückert Ina-Maria,Wichmann H-Erich,Azazi Dhoyazan,Plagnol Vincent,Pako Wandagi H.,Whitfield Jerome,Alpers Michael P.,Whittaker John,Balding David J.,Zerr Inga,Kretzschmar Hans,Collinge John

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/21/8/1897/17258635/ddr607.pdf

Reference40 articles.

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2. Molecular analysis of prion strain variation and the aetiology of ‘new variant’ CJD;Collinge;Nature,1996

3. Transmissions to mice indicate that ‘new variant’ CJD is caused by the BSE agent;Bruce;Nature,1997

4. The same prion strain causes vCJD and BSE;Hill;Nature,1997

5. Genetic predisposition to iatrogenic Creutzfeldt–Jakob disease;Collinge;Lancet,1991

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