Nf1+/−monocytes/macrophages induce neointima formation via CCR2 activation-Reference-Cited by-同舟云学术

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Nf1+/−monocytes/macrophages induce neointima formation via CCR2 activation

Published:2016-01-05 Issue:6 Volume:25 Page:1129-1139
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Hum. Mol. Genet.

Author:

Bessler Waylan K.,Kim Grace,Hudson Farlyn Z.,Mund Julie A.,Mali Raghuveer,Menon Keshav,Kapur Reuben,Clapp D. Wade,Ingram David A.,Stansfield Brian K.

Funder

Department of Defense

American Heart Association

Department of Pediatrics at Augusta University

National Institutes of Health

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/25/6/1129/7148409/ddv635.pdf

Reference65 articles.

1. Normal hematopoiesis and neurofibromin-deficient myeloproliferative disease require Erk

2. Cardiovascular disease in neurofibromatosis 1: Report of the NF1 Cardiovascular Task Force

3. Vascular abnormalities in patients with neurofibromatosis syndrome type I: Clinical spectrum, management, and results

4. Mortality in Neurofibromatosis 1: An Analysis Using U.S. Death Certificates

5. Cardiovascular malformations and other cardiovascular abnormalities in neurofibromatosis 1

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4. The Q181X Point Mutation in Nf1 Induces Cerebral Vessel Stenosis;Neuroscience Bulletin;2023-01-02

5. Childhood-Onset Refractory Hypertension Results from Neurofibromatosis Type 1 Caused by a Splicing <i>NF1</i> Mutation;Kidney and Blood Pressure Research;2023

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