PARK2 enhancement is able to compensate mitophagy alterations found in sporadic Alzheimer's disease-Reference-Cited by-同舟云学术

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PARK2 enhancement is able to compensate mitophagy alterations found in sporadic Alzheimer's disease

Published:2015-12-31 Issue:4 Volume:25 Page:792-806
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Hum. Mol. Genet.

Author:

Martín-Maestro Patricia,Gargini Ricardo,Perry George,Avila Jesús,García-Escudero Vega

Funder

Ministerio de Economía y Competitividad

National Institute on Minority Health and Health Disparities

National Institutes of Health

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/25/4/792/18527396/ddv616.pdf

Reference51 articles.

1. Age, Neuropathology, and Dementia

2. ALZHEIMER'S DISEASE: Genetic Studies and Transgenic Models

3. Abnormal mitochondrial dynamics in the pathogenesis of Alzheimer’s disease;Zhu;J. Alzheimers Dis.,2013

4. Oxidative stress in Alzheimer disease: A possibility for prevention

5. Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: Implications to mitochondria-targeted antioxidant therapeutics

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1. Disease-associated mutations in C-terminus of HSP70 interacting protein (CHIP) impair its ability to negatively regulate mitophagy;Neurobiology of Disease;2024-10

2. Effect of alternating nicotinamide phosphoribosyltransferase expression levels on mitophagy in Alzheimer's disease mouse models;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2024-10

3. AAV-aMTD-Parkin, a therapeutic gene delivery cargo, enhances motor and cognitive functions in Parkinson's and Alzheimer’s diseases;Pharmacological Research;2024-10

4. Translocator protein (TSPO) ligands attenuate mitophagy deficits in the SH-SY5Y cellular model of Alzheimer's disease via the autophagy adaptor P62;Biochimie;2024-09

5. Human fibroblasts from sporadic Alzheimer's disease (AD) patients show mitochondrial alterations and lysosome dysfunction;Free Radical Biology and Medicine;2024-09

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