TBX1 protein interactions and microRNA-96-5p regulation controls cell proliferation during craniofacial and dental development: implications for 22q11.2 deletion syndrome-Reference-Cited by-同舟云学术

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TBX1 protein interactions and microRNA-96-5p regulation controls cell proliferation during craniofacial and dental development: implications for 22q11.2 deletion syndrome

Published:2015-01-02 Issue:8 Volume:24 Page:2330-2348
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Gao S.,Moreno M.,Eliason S.,Cao H.,Li X.,Yu W.,Bidlack F. B.,Margolis H. C.,Baldini A.,Amendt B. A.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/24/8/2330/1841836/ddu750.pdf

Reference73 articles.

1. Velo-cardio-facial syndrome: A review of 120 patients

2. Chromosomal microdeletions: dissecting del22q11 syndrome

3. DiGeorge syndrome phenotype in mice mutant for the T-box gene, Tbx1

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1. Follistatin controls the number of murine teeth by limiting TGF-β signaling;iScience;2024-09

2. Exploring the roles of noncoding RNAs in craniofacial abnormalities: A systematic review;Developmental Biology;2024-01

3. Chromosome 22q11.2 Deletion Syndrome: A Comprehensive Review of Molecular Genetics in the Context of Multidisciplinary Clinical Approach;International Journal of Molecular Sciences;2023-05-05

4. Exploring microRNAs in craniofacial regenerative medicine;Biochemical Society Transactions;2023-04-19

5. CACNA1S mutation‐associated dental anomalies: A calcium channelopathy;Oral Diseases;2023-03-13

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