Huntingtin-associated protein 1 (Hap1) mutant mice bypassing the early postnatal lethality are neuroanatomically normal and fertile but display growth retardation-Reference-Cited by-同舟云学术

Huntingtin-associated protein 1 (Hap1) mutant mice bypassing the early postnatal lethality are neuroanatomically normal and fertile but display growth retardation

Published:2004-12-15 Issue:24 Volume:13 Page:3115-3125
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Dragatsis Ioannis,Zeitlin Scott,Dietrich Paula

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/13/24/3115/2090328/ddh328.pdf

Reference55 articles.

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2. Nasir, J., Maclean, A., Engelender, S., Duan, K., Margolis, R.L., Kleiderlein, J.J., Ross, C.A. and Hayden, M.R. (1999). Chromosomal localization of the Huntingtin associated protein (HAP-1) gene in mouse and humans with radiation hybrid and interspecific backcross mapping. Mamm Genome, 10, 397–398.

3. Block-Galarza, J., Chase, K.O., Sapp, E., Vaughn, K.T., Vallee, R.B., DiFiglia, M. and Aronin, N. (1997) Fast transport and retrograde movement of huntingtin and HAP 1 in axons. Neuroreport, 8, 2247–2251.

4. Li, S.H., Gutekunst, C.A., Hersch, S.M. and Li, X.J. (1998) Association of HAP1 isoforms with a unique cytoplasmic structure. J. Neurochem., 71, 2178–2185.

5. Torre, E.R., Coleman, S., Yi, H. and Gutekunst, C.A. (2003) A protocol for isolation and biochemical characterization of stigmoid bodies from rat brain. J. Neurosci. Methods, 125, 27–32.

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