HMGB1 Translocation After Ischemia in the Ovine Fetal Brain-Reference-Cited by-同舟云学术

HMGB1 Translocation After Ischemia in the Ovine Fetal Brain

Published:2016-05-05 Issue:6 Volume:75 Page:527-538
ISSN:0022-3069
Container-title:Journal of Neuropathology & Experimental Neurology
language:en
Short-container-title:J Neuropathol Exp Neurol

Author:

Zhang Jiyong,Klufas Daniel,Manalo Karina,Adjepong Kwame,Davidson , Joanne O.,Wassink Guido,Bennet Laura,Gunn Alistair J.,Stopa Edward G.,Liu Keyue,Nishibori Masahiro,Stonestreet Barbara S.

Publisher

Oxford University Press (OUP)

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Neurology,General Medicine,Pathology and Forensic Medicine

Link

http://academic.oup.com/jnen/article-pdf/75/6/527/7953593/nlw030.pdf

Reference71 articles.

1. Cooling for newborns with hypoxic ischaemic encephalopathy;Jacobs;Cochrane Database Syst Rev,2007

2. New pharmacologic and therapeutic approaches for hypoxic-ischemic encephalopathy in the newborn;Perrone;J Matern Fetal Neonatal Med,2012

3. Temperature profile and outcomes of neonates undergoing whole body hypothermia for neonatal hypoxic-ischemic encephalopathy

4. Childhood Outcomes after Hypothermia for Neonatal Encephalopathy

5. Neurobehavioral Assessment Predicts Motor Outcome in Preterm Infants

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