The chromosomal basis of species initiation: Prdm9 as an anti-speciation gene
Author:
Affiliation:
1. Department of Biomedical and Molecular Sciences, Queen’s University, Kingston, ON, Canada
Publisher
Oxford University Press (OUP)
Subject
Ecology, Evolution, Behavior and Systematics
Link
http://academic.oup.com/biolinnean/article-pdf/124/2/139/24954430/bly035.pdf
Reference66 articles.
1. A map of human PRDM9 binding provides evidence for novel behaviors of PRDM9 and other zinc-finger proteins in meiosis;Altemose;eLife,2017
2. Crossovers are associated with mutation and biased gene conversion at recombination hotspots;Arbeithuber;Proceedings of the National Academy of Sciences of the United States of America,2015
3. Multimer formation explains allelic suppression of PRDM9 recombination hotspots;Baker;PLoS Genetics,2015
4. Hybrid sterility locus on chromosome X controls meiotic recombination rate in mouse;Balcova;PLoS Genetics,2016
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1. Centenary of Haldane's ‘rule’: why male sterility may be normal, not ‘idiopathic’;Journal of Genetics;2022-05-10
2. PRDM9-directed recombination hotspots depleted near meiotically transcribed genes;Gene;2022-03
3. Revisiting George Romanes’ "Physiological Selection" (1886);Biological Theory;2020-07-20
4. When acting as a reproductive barrier for sympatric speciation, hybrid sterility can only be primary;Biological Journal of the Linnean Society;2019-10-04
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