Unveiling wild-type transthyretin cardiac amyloidosis as a significant and potentially modifiable cause of heart failure with preserved ejection fraction-Reference-Cited by-同舟云学术

Unveiling wild-type transthyretin cardiac amyloidosis as a significant and potentially modifiable cause of heart failure with preserved ejection fraction

Published:2015-07-28 Issue:38 Volume:36 Page:2595-2597
ISSN:0195-668X
Container-title:European Heart Journal
language:en
Short-container-title:Eur Heart J

Author:

Castaño Adam,Bokhari Sabahat,Maurer Mathew S.

Funder

NIH

Publisher

Oxford University Press (OUP)

Subject

Cardiology and Cardiovascular Medicine

Link

http://academic.oup.com/eurheartj/article-pdf/36/38/2595/7111199/ehv328.pdf

Reference16 articles.

1. Comorbidity and Ventricular and Vascular Structure and Function in Heart Failure With Preserved Ejection Fraction

2. A Novel Paradigm for Heart Failure With Preserved Ejection Fraction

3. Pressure-Volume Relationships in Patients With Transthyretin (ATTR) Cardiac Amyloidosis Secondary to V122I Mutations and Wild-Type Transthyretin

4. Guidelines on the management of valvular heart disease (version 2012)

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