Channelopathies, cardiac hypertrophy, and the theory of light
Author:
Affiliation:
1. Cardiomyopathy Unit, Careggi University Hospital, Florence, Italy
2. Department NeuroFarBa, University of Florence, Italy
Funder
Italian Ministry of Health
Telethon Italy
ToRSADE
Publisher
Oxford University Press (OUP)
Subject
Cardiology and Cardiovascular Medicine
Link
http://academic.oup.com/eurheartj/article-pdf/39/31/2908/25515589/ehy297.pdf
Reference15 articles.
1. A common co-morbidity modulates disease expression and treatment efficacy in inherited cardiac sodium channelopathy;Rivaud;Eur Heart J,2018
2. Fibrosis, connexin-43, and conduction abnormalities in the Brugada syndrome;Nademanee;J Am Coll Cardiol,2015
3. Progression of electroanatomic substrate and electric storm recurrence in a patient with Brugada syndrome;Notarstefano;Circulation,2015
4. Patient-specific and genome-edited induced pluripotent stem cell-derived cardiomyocytes elucidate single-cell phenotype of Brugada syndrome;Liang;J Am Coll Cardiol,2016
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