Regulation of osteoclast apoptosis by ubiquitylation of proapoptotic BH3-only Bcl-2 family member Bim
Author:
Publisher
Wiley
Subject
General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience
Reference54 articles.
1. Insulin receptor substrate-2 maintains predominance of anabolic function over catabolic function of osteoblasts
2. Molecular mechanisms of bone resorption by the osteoclast
3. Nerve Growth Factor (NGF) Down-regulates the Bcl-2 Homology 3 (BH3) Domain-only Protein Bim and Suppresses Its Proapoptotic Activity by Phosphorylation
4. Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity
5. BH3-only Bcl-2 family member Bim is required for apoptosis of autoreactive thymocytes
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