Homozygous FCGR3A-158F mutation is associated with delayed B-cell depletion following rituximab but with preserved efficacy in a patient with refractory lupus nephritis-Reference-Cited by-同舟云学术

Homozygous FCGR3A-158F mutation is associated with delayed B-cell depletion following rituximab but with preserved efficacy in a patient with refractory lupus nephritis

Published:2012-12-20 Issue:1 Volume:6 Page:74-76
ISSN:2048-8505
Container-title:Clinical Kidney Journal
language:en
Short-container-title:Clin Kidney J

Author:

Seret Guillaume,Hanrotel Catherine,Bendaoud Boutahar,Le Meur Yannick,Renaudineau Yves

Publisher

Oxford University Press (OUP)

Subject

Transplantation,Nephrology

Link

http://academic.oup.com/ckj/article-pdf/6/1/74/1270549/sfs162.pdf

Reference10 articles.

1. Remission of refractory lupus nephritis with a protocol including rituximab

2. Efficacy and safety of rituximab in patients with active proliferative lupus nephritis: The lupus nephritis assessment with rituximab study

3. BAFF-modulated repopulation of B lymphocytes in the blood and salivary glands of RITUXIMAB-TREATED patients with Sjögren's syndrome

4. Baseline autoantibody profiles predict normalization of complement and anti-dsDNA autoantibody levels following Rituximab treatment in systemic lupus erythematosus

5. B cell depletion therapy in systemic lupus erythaematosus: relationships among serum B lymphocyte stimulator levels, autoantibody profile and clinical response

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