Trimetazidine inhibits pressure overload-induced cardiac fibrosis through NADPH oxidase–ROS–CTGF pathway-Reference-Cited by-同舟云学术

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Trimetazidine inhibits pressure overload-induced cardiac fibrosis through NADPH oxidase–ROS–CTGF pathway

Published:2010-06-09 Issue:1 Volume:88 Page:150-158
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Liu Xuehui,Gai Yingli,Liu Fei,Gao Wei,Zhang Youyi,Xu Ming,Li Zhaoping

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/88/1/150/17390596/cvq181.pdf

Reference41 articles.

1. Myocardial fibrosis: functional significance and regulatory factors;Weber;Cardiovasc Res,1993

2. Myocardial stiffness is determined by ventricular fibrosis, but not by compensatory or excessive hypertrophy in hypertensive heart;Yamamoto;Cardiovasc Res,2002

3. Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms;Hein;Circulation,2003

4. The antianginal drug trimetazidine shifts cardiac energy metabolism from fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-ketoacyl coenzyme A thiolase;Kantor;Circ Res,2000

5. Beneficial effects of trimetazidine in ex vivo working ischemic hearts are due to a stimulation of glucose oxidation secondary to inhibition of long-chain 3-ketoacyl coenzyme a thiolase;Lopaschuk;Circ Res,2003

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2. The Antioxidant Properties and Cardiovascular, Antidiabetic, and Renal Effects of Trimetazidine Described in Research on Experimental Models;Current Reviews in Clinical and Experimental Pharmacology;2024-08-20

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4. Trimetazidine Attenuates Ischemia/Reperfusion-Induced Myocardial Ferroptosis by Modulating the Sirt3/Nrf2-GSH System and Reducing Mitochondrial Oxidative Stress;2023-10-18

5. Trimetazidine to Reduce Myocardial Fibrosis—Competing Interests with SGLT2 Inhibitors?;Cardiovascular Drugs and Therapy;2023-09-13

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