Poly(A) binding protein nuclear 1 levels affect alternative polyadenylation-Reference-Cited by-同舟云学术

Poly(A) binding protein nuclear 1 levels affect alternative polyadenylation

Published:2012-07-05 Issue:18 Volume:40 Page:9089-9101
ISSN:1362-4962
Container-title:Nucleic Acids Research
language:en
Short-container-title:

Author:

de Klerk Eleonora¹,Venema Andrea¹,Anvar S. Yahya¹,Goeman Jelle J.¹,Hu OuHua¹,Trollet Capucine¹¹,Dickson George¹,den Dunnen Johan T.¹,van der Maarel Silvère M.¹,Raz Vered¹,‘t Hoen Peter A. C.¹

Affiliation:

1. Center for Human and Clinical Genetics, 2 Department of Medical Statistics and Bioinformatics, Leiden University Medical Center, 2300 RC Leiden, The Netherlands, 3 School of Biological Sciences, Royal Holloway – University of London, Egham, Surrey, TW20 0EX, UK and 4 Thérapie des Maladies du Muscle Strié, Institut de Myologie, UM76, Université Pierre et Marie Curie, INSERM-U974, CNRS-UMR7215, Paris, France

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/article-pdf/40/18/9089/25348359/gks655.pdf

Reference53 articles.

1. Loss of nuclear poly(A)-binding protein 1 causes defects in myogenesis and mRNA biogenesis;Apponi;Hum. Mol. Genet.,2010

2. Poly(A) tail length is controlled by the nuclear poly(A)-binding protein regulating the interaction between poly(A) polymerase and the cleavage and polyadenylation specificity factor;Kuhn;J. Biol. Chem.,2009

3. Poly(A) tail length control is caused by termination of processive synthesis;Wahle;J. Biol. Chem.,1995

4. An essential cytoplasmic function for the nuclear poly(A) binding protein, PABP2, in poly(A) tail length control and early development in Drosophila;Benoit;Dev. Cell,2005

5. Short GCG expansions in the PABP2 gene cause oculopharyngeal muscular dystrophy;Brais;Nat. Genet.,1998

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