Interleukin-18 Inhibition in Inflammatory Bowel Disease: A Delicate Balance-Reference-Cited by-同舟云学术

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Interleukin-18 Inhibition in Inflammatory Bowel Disease: A Delicate Balance

Published:2024-03-05 Issue:4 Volume:30 Page:693-694
ISSN:1078-0998
Container-title:Inflammatory Bowel Diseases
language:en
Short-container-title:

Author:

Geertsema Sem¹,Fagundes Raphael R²,Otten Antonius T¹,Dijkstra Gerard¹,Faber Klaas Nico¹,Bourgonje Arno R¹³^ORCID

Affiliation:

1. Department of Gastroenterology and Hepatology, University Medical Center Groningen, University of Groningen , Groningen , the Netherlands

2. Department of Cell and Chemical Biology, Leiden University Medical Center , Leiden , the Netherlands

3. Henry D. Janowitz Division of Gastroenterology, Department of Medicine, Icahn School of Medicine at Mount Sinai , New York, NY , United States

Publisher

Oxford University Press (OUP)

Link

https://academic.oup.com/ibdjournal/advance-article-pdf/doi/10.1093/ibd/izae042/56873607/izae042.pdf

Reference10 articles.

1. Monoclonal antibody against mature interleukin-18 ameliorates colitis in mice and improves epithelial barrier function;Ikegami;Inflamm Bowel Dis.,2023

2. Epithelial-derived IL-18 regulates Th17 cell differentiation and Foxp3+ Treg cell function in the intestine;Harrison;Mucosal Immunol,2015

3. Microbiota-modulated metabolites shape the intestinal microenvironment by regulating NLRP6 inflammasome signaling;Levy;Cell.,2015

4. Epithelial IL-18 equilibrium controls barrier function in colitis;Nowarski;Cell.,2015

5. Dual roles of IL-18 in colitis through regulation of the function and quantity of goblet cells;Pu;Int J Mol Med.,2019

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Electroacupuncture at Zusanli regulates the pathological phenotype of inflammatory bowel disease by modulating the NLRP3 inflammasome pathway;Immunity, Inflammation and Disease;2024-08

2. Reply: Interleukin-18 Inhibition in Inflammatory Bowel Diseases: A Delicate Balance;Inflammatory Bowel Diseases;2024-03-05

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