Quantitative analysis of γH2AX reveals distinct responses in multiple mouse organs after administration of mitomycin C or ethyl methanesulfonate
Author:
Affiliation:
1. Safety Evaluation Center, Ecology and Quality Management Division, CSR Division, FUJIFILM Corporation, 210 Nakanuma, Minamiashigara, Kanagawa, Japan
Publisher
Oxford University Press (OUP)
Subject
Health, Toxicology and Mutagenesis,Genetics(clinical),Toxicology,Genetics
Link
http://academic.oup.com/mutage/article-pdf/33/5-6/371/27698059/gey040.pdf
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3. MDC1 maintains genomic stability by participating in the amplification of ATM-dependent DNA damage signals;Lou;Mol. Cell,2006
4. DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139;Rogakou;J. Biol. Chem,1998
5. Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses;Rothkamm;Proc. Natl. Acad. Sci. USA,2003
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1. Validation of the γH2AX biomarker for genotoxicity assessment: a review;Archives of Toxicology;2019-07-09
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