Early host cell reactivation of an oxidatively damaged adenovirus-encoded reporter gene requires the Cockayne syndrome proteins CSA and CSB
Author:
Publisher
Oxford University Press (OUP)
Subject
Health, Toxicology and Mutagenesis,Genetics(clinical),Toxicology,Genetics
Link
http://academic.oup.com/mutage/article-pdf/26/2/315/3818877/geq096.pdf
Reference38 articles.
1. Mechanisms of Formation, Genotoxicity, and Mutation of Guanine Oxidation Products
2. Assay of excised oxidative DNA lesions: isolation of 8-oxoguanine and its nucleoside derivatives from biological fluids with a monoclonal antibody column.
3. Base-excision repair of oxidative DNA damage
4. Repair of 8-oxoguanine in DNA is deficient in Cockayne syndrome group B cells
5. A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous oxidative DNA base damage
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Development of a screening system for DNA damage and repair of potential carcinogens based on dual luciferase assay in human HepG2 cell;Mutagenesis;2013-06-22
2. Host cell reactivation of gene expression for an adenovirus-encoded reporter gene reflects the repair of UVC-induced cyclobutane pyrimidine dimers and methylene blue plus visible light-induced 8-oxoguanine;Mutagenesis;2013-06-21
3. Reduced host cell reactivation of oxidatively damaged DNA in ageing human fibroblasts;Oncology Reports;2013-03-22
4. Mfd Is Required for Rapid Recovery of Transcription following UV-Induced DNA Damage but Not Oxidative DNA Damage in Escherichia coli;Journal of Bacteriology;2012-03-16
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