Dexrazoxane mitigates epirubicin-induced genotoxicity in mice bone marrow cells-Reference-Cited by-同舟云学术

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Dexrazoxane mitigates epirubicin-induced genotoxicity in mice bone marrow cells

Published:2015-09-22 Issue:2 Volume:31 Page:137-145
ISSN:0267-8357
Container-title:Mutagenesis
language:en
Short-container-title:MUTAGE

Author:

Attia Sabry M.,Ahmad Sheikh F.,Saquib Quaiser,Harisa Gamaleldin I.,Al-Khedhairy Abdulaziz A.,Bakheet Saleh A.

Publisher

Oxford University Press (OUP)

Subject

Health, Toxicology and Mutagenesis,Genetics (clinical),Toxicology,Genetics

Link

http://academic.oup.com/mutage/article-pdf/31/2/137/8178243/gev065.pdf

Reference49 articles.

1. Anthracyclines: Molecular Advances and Pharmacologic Developments in Antitumor Activity and Cardiotoxicity

2. Anthracyclines in the treatment of cancer. An overview;Hortobágyi;Drugs,1997

3. A critical evaluation of the mechanisms of action proposed for the antitumor effects of the anthracycline antibiotics adriamycin and daunorubicin

4. Selected pharmacologic characteristics of idarubicin and idarubicinol;Ames;Leukemia,1992

5. Mutagenicity of some topoisomerase II-interactive agents;Attia;Saudi Pharm. J,2008

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Guidance for the use and interpretation of assays for monitoring anti-genotoxicity;Life Sciences;2024-01

2. Investigation of belinostat-induced genomic instability by molecular cytogenetic analysis and pathway-focused gene expression profiling;Toxicology and Applied Pharmacology;2018-07

3. Modulatory Effects of Dexrazoxane Against Genotoxicity and Lipid Peroxidation Induced by Idarubicin in HepG2 Cells;International Journal of Cancer Management;2018-01-14

4. Dexrazoxane Averts Idarubicin-Evoked Genomic Damage by Regulating Gene Expression Profiling Associated With the DNA Damage-Signaling Pathway in BALB/c Mice;Toxicological Sciences;2017-08-16

5. Utility of Dexrazoxane for the Attenuation of Epirubicin-Induced Genetic Alterations in Mouse Germ Cells;PLOS ONE;2016-09-30

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