Severe B cell deficiency and disrupted splenic architecture in transgenic mice expressing the E41K mutated form of Bruton's tyrosine kinase-Reference-Cited by-同舟云学术

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Severe B cell deficiency and disrupted splenic architecture in transgenic mice expressing the E41K mutated form of Bruton's tyrosine kinase

Published:1998-09-15 Issue:18 Volume:17 Page:5309-5320
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Dingjan Gemma M.,Maas Alex,Nawijn Martijn C.,Smit Linda,Voerman Jane S.A.,Grosveld Frank,Hendriks Rudolf W.

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Cited by 53 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. E41K mutation activates Bruton’s tyrosine kinase by stabilizing an inositol hexakisphosphate-dependent invisible dimer;Journal of Biological Chemistry;2024-08

2. E41K Mutation Activates Bruton’s Tyrosine Kinase by Stabilizing an Inositol Hexakisphosphate dependent Invisible Dimer;2024-01-23

3. Bruton’s Tyrosine Kinase-Mediated Signaling in Myeloid Cells Is Required for Protective Innate Immunity During Pneumococcal Pneumonia;Frontiers in Immunology;2021-09-06

4. MOG autoantibodies trigger a tightly-controlled FcR and BTK-driven microglia proliferative response;Brain;2021-06-19

5. Map3k14 as a Regulator of Innate and Adaptive Immune Response during Acute Viral Infection;Pathogens;2020-02-04

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