Hypokalaemia and paralysis in the Thai population
Author:
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/19/8/2013/5410451/gfh328.pdf
Reference17 articles.
1. Dihydropyridine receptor mutations cause hypokalemic periodic paralysis
2. Mapping of the hypokalaemic periodic paralysis (HypoPP) locus to chromosome 1q31–32 in three European families
3. Identification of mutations in the CACNL1A3 gene in 13 families of Scandinavian origin having hypokalemic periodic paralysis and evidence of a founder effect in Danish families
4. Voltage-sensor sodium channel mutations cause hypokalemic periodic paralysis type 2 by enhanced inactivation and reduced current
5. A novel sodium channel mutation in a family with hypokalemic periodic paralysis
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1. Nephrology in Thailand;Nephrology Worldwide;2021
2. Impact of first-line treatment choice on long-term outcomes of hyperthyroid Graves’ disease patients with thyrotoxic periodic paralysis;Journal of Clinical & Translational Endocrinology;2020-09
3. Periodic Paralysis and Encephalopathy as Initial Manifestations of Graves’ Disease;The Neurologist;2017-07
4. Association of KCNJ2 Genetic Variants with Susceptibility to Thyrotoxic Periodic Paralysis in Patients with Graves’ Disease;Experimental and Clinical Endocrinology & Diabetes;2016-12-22
5. Thyrotoxic Periodic Paralysis and Polymorphisms of the ADRB2, AR, and GABRA3 Genes in Men with Graves Disease;Endocrinology and Metabolism;2016
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