Regulation of B cell development and B cell signalling by CD22 and its ligands α2,6-linked sialic acids
Author:
Publisher
Oxford University Press (OUP)
Subject
Immunology,General Medicine,Immunology and Allergy
Link
http://academic.oup.com/intimm/article-pdf/18/4/603/1879496/dxh402.pdf
Reference39 articles.
1. Nitschke, L. 2005. The role of CD22 and other inhibitory co-receptors in B-cell activation. Curr. Opin. Immunol.17:290.
2. Doody, G. M., Justement, L. B., Delibrias et al.1995. A role in B cell activation for CD22 and the protein tyrosine phosphatase SHP. Science269:242.
3. Sato, S., Jansen, P. J. and Tedder, T. F. 1997. CD19 and CD22 expression reciprocally regulates tyrosine phosphorylation of Vav protein during B lymphocyte signaling. Proc. Natl Acad. Sci. USA94:13158.
4. Gerlach, J., Ghosh, S., Jumaa, H. et al.2003. B cell defects in SLP65/BLNK-deficient mice can be partially corrected by the absence of CD22, an inhibitory coreceptor for BCR signaling. Eur. J. Immunol.33:3418.
5. Chen, J., McLean, P. A., Neel, B. G., Okunade, G., Shull, G. E. and Wortis, H. H. 2004. CD22 attenuates calcium signaling by potentiating plasma membrane calcium-ATPase activity. Nat. Immunol.5:651.
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