Endogenous IL-33 enhances Th2 cytokine production and T-cell responses during allergic airway inflammation-Reference-Cited by-同舟云学术

Endogenous IL-33 enhances Th2 cytokine production and T-cell responses during allergic airway inflammation

Published:2011-03-21 Issue:5 Volume:23 Page:307-315
ISSN:1460-2377
Container-title:International Immunology
language:en
Short-container-title:

Author:

Louten Jennifer,Rankin Andrew L.,Li Ying,Murphy Erin E.,Beaumont Maribel,Moon Christina,Bourne Patricia,McClanahan Terrill K.,Pflanz Stefan,de Waal Malefyt Rene

Publisher

Oxford University Press (OUP)

Subject

Immunology,General Medicine,Immunology and Allergy

Link

http://academic.oup.com/intimm/article-pdf/23/5/307/17414361/dxr006.pdf

Reference31 articles.

1. Suppression of interleukin-33 bioactivity through proteolysis by apoptotic caspases;Luthi;Immunity,2009

2. The IL-1-like cytokine IL-33 is inactivated after maturation by caspase-1;Cayrol;Proc. Natl. Acad. Sci. USA,2009

3. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines;Schmitz;Immunity,2005

4. IL-1 receptor accessory protein and ST2 comprise the IL-33 receptor complex;Chackerian;J. Immunol.,2007

5. Anti-IL-33 antibody treatment inhibits airway inflammation in a murine model of allergic asthma;Liu;Biochem. Biophys. Res. Commun.,2009

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