Roles of IL-22 in allergic airway inflammation in mice and humans

Author:

Hirose Koichi12,Ito Takashi1,Nakajima Hiroshi1

Affiliation:

1. Department of Allergy and Clinical Immunology, Graduate School of Medicine, Chiba University, Chuo-ku, Chiba, Japan

2. Department of Rheumatology, School of Medicine, International University of Health and Welfare, Narita City, Chiba, Japan

Funder

Ministry of Education, Culture, Sports, Science and Technology

Publisher

Oxford University Press (OUP)

Subject

Immunology,General Medicine,Immunology and Allergy

Reference70 articles.

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2. Type 2 inflammation in asthma—present in most, absent in many;Fahy;Nat. Rev. Immunol,2015

3. Predominant TH2-like bronchoalveolar T-lymphocyte population in atopic asthma;Robinson;N. Engl. J. Med,1992

4. IL-4 and IL-5 mRNA and protein in bronchial biopsies from patients with atopic and nonatopic asthma: evidence against ‘intrinsic’ asthma being a distinct immunopathologic entity;Humbert;Am. J. Respir. Crit. Care Med,1996

5. Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model;Foster;J. Exp. Med,1996

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