A mathematical model-based analysis of the time–kill kinetics of ceftazidime/avibactam against Pseudomonas aeruginosa
Author:
Affiliation:
1. Department of Pharmaceutics, College of Pharmacy, University of Florida, Gainesville, FL, USA
2. Department of Molecular Genetics and Microbiology, College of Medicine, University of Florida, Gainesville, FL, USA
3. AstraZeneca, Waltham, MA, USA
Funder
AstraZeneca
Publisher
Oxford University Press (OUP)
Subject
Infectious Diseases,Pharmacology (medical),Pharmacology,Microbiology (medical)
Link
http://academic.oup.com/jac/article-pdf/73/5/1295/24609986/dkx537.pdf
Reference35 articles.
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3. Avibactam and class C β-lactamases: mechanism of inhibition, conservation of the binding pocket, and implications for resistance;Lahiri;Antimicrob Agents Chemother,2014
4. Molecular basis of selective inhibition and slow reversibility of avibactam against class D carbapenemases: a structure-guided study of OXA-24 and OXA-48;Lahiri;ACS Chem Biol,2015
5. Structural and sequence analysis of class A β-lactamases with respect to avibactam inhibition: impact of Omega-loop variations;Lahiri;J Antimicrob Chemother,2016
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