Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system-Reference-Cited by-同舟云学术

Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system

Published:2017-10-12 Issue:8 Volume:75 Page:
ISSN:2049-632X
Container-title:Pathogens and Disease
language:en
Short-container-title:

Author:

Horridge Danielle N¹,Begley Allison A¹²,Kim June¹,Aravindan Neeraja¹,Fan Kexin¹,Forsyth Mark H¹

Affiliation:

1. Department of Biology, The College of William and Mary, Williamsburg VA 23187, USA

2. The Governor's School of Science and Technology. Hampton, VA 23666, USA

Funder

National Institute of Allergy and Infectious Diseases

American Society for Microbiology Undergraduate Research

Publisher

Oxford University Press (OUP)

Subject

Infectious Diseases,Microbiology (medical),General Immunology and Microbiology,General Medicine,Immunology and Allergy

Link

http://academic.oup.com/femspd/article-pdf/75/8/ftx113/28039707/ftx113.pdf

Reference41 articles.

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3. Type IV secretion and signal transduction of Helicobacter pylori CagA through interactions with host cell receptors;Backert;Toxins (Basel),2017

4. Composition, structure and function of the Helicobacter pylori cag pathogenicity island encoded type IV secretion system;Backert;Future Microbiol,2015

5. Therapeutic vaccination with Salmonella-derived codon optimized outer inflammatory protein DNA vaccine enhances protection in Helicobacter pylori infected mice;Chen;Vaccine,2012

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