The pyrin inflammasome: from sensing RhoA GTPases-inhibiting toxins to triggering autoinflammatory syndromes-Reference-Cited by-同舟云学术

The pyrin inflammasome: from sensing RhoA GTPases-inhibiting toxins to triggering autoinflammatory syndromes

Published:2018-03-28 Issue:3 Volume:76 Page:
ISSN:2049-632X
Container-title:Pathogens and Disease
language:en
Short-container-title:

Author:

Jamilloux Yvan¹²,Magnotti Flora¹,Belot Alexandre¹³,Henry Thomas¹^ORCID

Affiliation:

1. Centre International de Recherche en Infectiologie (CIRI), Inserm U1111, Université Claude Bernard Lyon 1, CNRS, UMR5308, Ecole Normale Supérieure de Lyon, F-69007 Lyon, France

2. Departement de Médecine Interne, Hopital de la Croix-Rousse, Université Claude Bernard Lyon 1, F-69004 Lyon, France

3. Service de Néphrologie, Rhumatologie, Dermatologie pédiatrique, Hôpital Femme Mère Enfant, Hospices Civils de Lyon, F-69677 Lyon, France

Publisher

Oxford University Press (OUP)

Subject

Infectious Diseases,Microbiology (medical),General Immunology and Microbiology,General Medicine,Immunology and Allergy

Link

http://academic.oup.com/femspd/article-pdf/76/3/fty020/24773521/fty020.pdf

Reference91 articles.

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3. Identification of a putative target for Rho as the serine-threonine kinase protein kinase N;Amano;Science,1996

4. EspM2 is a RhoA guanine nucleotide exchange factor;Arbeloa;Cell Microbiol,2010

5. A burkholderia type VI effector deamidates Rho GTPases to activate the pyrin inflammasome and trigger inflammation;Aubert;Cell Host Microbe,2016

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