The role of Schizosaccharomyces pombe Rad32, the Mre11 homologue, and other DNA damage response proteins in non-homologous end joining and telomere length maintenance-Reference-Cited by-同舟云学术

The role of Schizosaccharomyces pombe Rad32, the Mre11 homologue, and other DNA damage response proteins in non-homologous end joining and telomere length maintenance

Published:1999-07-01 Issue:13 Volume:27 Page:2655-2661
ISSN:0305-1048
Container-title:Nucleic Acids Research
language:en
Short-container-title:Nucleic Acids Research

Author:

Wilson S.,Warr N.,Taylor D. L.,Watts F. Z.

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/article-pdf/27/13/2655/7042734/27-13-2655.pdf

Reference57 articles.

1. Two different types of double-strand breaks in Saccharomyces cerevisiae are repaired by similar RAD52-independent, nonhomologous recombination events.

2. A modified single-strand annealing model best explains the joining of DNA double-strand breaks in mammalian cells and cell extracts

3. Characterization of recombination intermediates from DNA injected into Xenopus laevis oocytes: evidence for a nonconservative mechanism of homologous recombination.

4. Two alternative pathways of double-strand break repair that are kinetically separable and independently modulated.

5. Test of the double-strand-break repair model of recombination in Xenopus laevis oocytes.

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