Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function-Reference-Cited by-同舟云学术

Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function

Published:2001-05 Issue:5 Volume:2 Page:438-445
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Jones Nina¹²,Voskas Daniel¹²,Master Zubin¹²,Sarao Renu³,Jones Jamie²,Dumont Daniel J¹²⁴

Affiliation:

1. Department of Medical Biophysics, University of Toronto Toronto Ontario Canada

2. Molecular and Cellular Biology Research, Sunnybrook and Women's College Health Sciences Centre 2075 Bayview Avenue, Research Building, S‐227 Toronto Ontario Canada M4N 3M5

3. Amgen Institute Toronto Ontario Canada M5G 2C1

4. Toronto‐Sunnybrook Regional Cancer Centre Toronto Ontario Canada

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1093/embo-reports/kve093

Reference20 articles.

1. Isolation of Angiopoietin-1, a Ligand for the TIE2 Receptor, by Secretion-Trap Expression Cloning

2. tek, a novel tyrosine kinase gene located on mouse chromosome 4, is expressed in endothelial cells and their presumptive precursors;Dumont D.J.;Oncogene,1992

3. Dominant-negative and targeted null mutations in the endothelial receptor tyrosine kinase, tek, reveal a critical role in vasculogenesis of the embryo.

4. Vascularization of the mouse embryo: A study of flk-1, tek, tie, and vascular endothelial growth factor expression during development

5. Blood Vessel Formation: What Is Its Molecular Basis?

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