An intact NEDD8 pathway is required for Cullin‐dependent ubiquitylation in mammalian cells-Reference-Cited by-同舟云学术

An intact NEDD8 pathway is required for Cullin‐dependent ubiquitylation in mammalian cells

Published:2002-02 Issue:2 Volume:3 Page:177-182
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Ohh Michael¹,Kim William Y¹,Moslehi Javid J¹,Chen Yuzhi²,Chau Vincent³,Read Margaret A⁴,Kaelin William G¹⁵

Affiliation:

1. Department of Adult Oncology, Dana‐Farber Cancer Institute and Brigham and Women's Hospital, Harvard Medical School Boston MA 02115 USA

2. Department of Psychiatry, Harvard Medical School, McLean Hospital Belmont MA 02478 USA

3. Department of Cellular and Molecular Physiology, Milton S. Hershey Medical Center, Pennsylvania State University College of Medicine Hershey PA 17033 USA

4. Millenium Pharmaceuticals Cambridge MA 02139 USA

5. Howard Hughes Medical Institute Chevy Chase MD 20815 USA

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1093/embo-reports/kvf028

Reference41 articles.

1. The Amyloid Precursor Protein-binding Protein APP-BP1 Drives the Cell Cycle through the S-M Checkpoint and Causes Apoptosis in Neurons

2. Accumulation of p53 in a mutant cell line defective in the ubiquitin pathway.

3. Ubiquitin-mediated proteolysis: biological regulation via destruction

4. The Arabidopsis cullin AtCUL1 is modified by the ubiquitin-related protein RUB1

5. Suppressor of Cytokine Signaling-1 Inhibits VAV Function through Protein Degradation

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