PLK1 targets CtIP to promote microhomology-mediated end joining-Reference-Cited by-同舟云学术

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PLK1 targets CtIP to promote microhomology-mediated end joining

Published:2018-09-08 Issue: Volume: Page:
ISSN:0305-1048
Container-title:Nucleic Acids Research
language:en
Short-container-title:

Author:

Wang Hailong¹^ORCID,Qiu Zhiyu¹,Liu Bo¹,Wu Yan¹,Ren Jianping¹,Liu Yaqing¹,Zhao Yuqin¹,Wang Ya¹,Hao Shuailin¹,Li Zheng¹,Peng Bin²,Xu Xingzhi²

Affiliation:

1. Beijing Key Laboratory of DNA Damage Response and College of Life Sciences, Capital Normal University, Beijing 100048, China

2. Guangdong Key Laboratory for Genome Stability & Disease Prevention, Shenzhen University School of Medicine, Shenzhen, Guangdong 518060, China

Funder

National Basic Research Program of China

National Natural Science Foundation of China

Beijing Natural Science Foundation

Shenzhen Science and Technology Innovation Commission

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/advance-article-pdf/doi/10.1093/nar/gky810/25699014/gky810.pdf

Reference67 articles.

1. The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway;Lieber;Annu. Rev. Biochem.,2010

2. Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis;Moynahan;Nat. Rev. Mol. Cell Biol.,2010

3. Mechanism of eukaryotic homologous recombination;San Filippo;Annu. Rev. Biochem.,2008

4. Double-strand break end resection and repair pathway choice;Symington;Annu. Rev. Genet.,2011

5. Microhomology-mediated end joining: new players join the team;Wang;Cell Biosci.,2017

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