Long-term dehydroepiandrosterone and 16 -fluoro-5-androsten-17-one administration enhances DNA synthesis and induces expression of c-fos and c-Ha-ras in a selected population of preneoplastic lesions in liver of diethylnitrosamine-initiated rats-Reference-Cited by-同舟云学术

Long-term dehydroepiandrosterone and 16 -fluoro-5-androsten-17-one administration enhances DNA synthesis and induces expression of c-fos and c-Ha-ras in a selected population of preneoplastic lesions in liver of diethylnitrosamine-initiated rats

Published:2001-02-01 Issue:2 Volume:22 Page:301-308
ISSN:0143-3334
Container-title:Carcinogenesis
language:en
Short-container-title:Carcinogenesis

Author:

Simile M. M.,De Miglio M. R.,Calvisi D.,Muroni M. R.,Frau M.,Asara G.,Daino L.,Deiana L.,Pascale R. M.,Feo F.

Publisher

Oxford University Press (OUP)

Subject

Cancer Research,General Medicine

Link

http://academic.oup.com/carcin/article-pdf/22/2/301/9417715/0220301.pdf

Reference46 articles.

1. Sex-dependent, tissue-specific opposing effects of dehydroepiandrosterone on initiation and modulation stages of liver and lung carcinogenesis induced by dihydroxy-di-n-propylnitrosamine in F344 rats

2. Inhibition by Dehydroepiandrosterone of Liver Preneoplastic Foci Formation in Rats After Initiation-Selection in Experimental Carcinogenesis

3. Reversal by ribo- and deoxyribonucleosides of dehydroepiandrosterone-induced inhibition of enzyme altered foci in the liver of rats subjected to the initiation-selection process of experimental carcinogenesis

4. Phenotypic modulation of hepatocarcinogenesis and reduction in N-nitrosomorpholine-induced hemangiosarcoma and adrenal lesion development in Sprague-Dawley rats by dehydroepiandrosterone

5. Inhibition by dehydroepiandrosterone of growth and progression of persistent liver nodules in experimental rat liver carcinogenesis

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