SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic-Reference-Cited by-同舟云学术

SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic

Published:2012-02-27 Issue:3 Volume:94 Page:450-459
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Boink Gerard J.J.,Lau David H.,Shlapakova Iryna N.,Sosunov Eugene A.,Anyukhovsky Evgeny P.,Driessen Helen E.,Dun Wen,Chen Ming,Danilo Peter,Rosen Tove S.,Őzgen Nazira,Duffy Heather S.,Kryukova Yelena,Boyden Penelope A,Robinson Richard B.,Brink Peter R.,Cohen Ira S.,Rosen Michael R.

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/94/3/450/17236672/cvs107.pdf

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4. Rotigaptide (zp123) prevents spontaneous ventricular arrhythmias and reduces infarct size during myocardial ischemia/reperfusion injury in open-chest dogs;Hennan;J Pharmacol Exp Ther,2006

5. Characterization of gap junction remodeling in epicardial border zone of healing canine infarcts and electrophysiological effects of partial reversal by rotigaptide;Macia;Circ Arrhythm Electrophysiol,2011

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