Peripheral nerve pathology, including aberrant Schwann cell differentiation, is ameliorated by doxycycline in a laminin- 2-deficient mouse model of congenital muscular dystrophy-Reference-Cited by-同舟云学术

Peripheral nerve pathology, including aberrant Schwann cell differentiation, is ameliorated by doxycycline in a laminin- 2-deficient mouse model of congenital muscular dystrophy

Published:2011-04-19 Issue:13 Volume:20 Page:2662-2672
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Homma S.,Beermann M. L.,Miller J. B.

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Reference53 articles.

1. Distribution and Function of Laminins in the Neuromuscular System of Developing, Adult, and Mutant Mice

2. A single point mutation in the LN domain of LAMA2 causes muscular dystrophy and peripheral amyelination

3. The congenital muscular dystrophies in 2004: a century of exciting progress

4. Congenital muscular dystrophy. Part I: a review of phenotypical and diagnostic aspects

5. Congenital Muscular Dystrophies: Toward Molecular Therapeutic Interventions

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2. LAMA2 Neuropathies: Human Findings and Pathomechanisms From Mouse Models;Frontiers in Molecular Neuroscience;2020-04-23

3. A Family of Laminin α2 Chain-Deficient Mouse Mutants: Advancing the Research on LAMA2-CMD;Frontiers in Molecular Neuroscience;2020-04-21

4. A mutation-independent approach for muscular dystrophy via upregulation of a modifier gene;Nature;2019-07-24

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